Tuesday, December 24, 2024

Biological Approach to Chronic Lyme Disease

 

Lyme disease is an insect-borne illness caused by the bacteria Borrelia burgdorferi. It is the most common vector borne disease in North America and Europe, with 300,000 new cases in the U.S. and 100,000 new cases in Europe reported annually. These numbers are likely underestimated as most infections go undiagnosed. The Lyme bacterium Borrelia is a spirochete that moves quickly through tissues like a corkscrew. If one were to measure the time it would take an injected Borrelia bacterium to travel from the tongue to the soles of the feet, it would take only a matter of seconds. It can move 2000 times faster than the white blood cells dispatched by the body's immune system to catch it. It is that efficient at drilling through connective tissue, using the body's own enzyme chemistry to soften tissue.

In the last 40 years people in the West have been suffering from a severely weaponized version of Lyme disease since the 1970's – some may suffer in bed with significant pain for years, often not able to even raise a brow. Very sad, with no apparent remedy. This version is increasingly found in those suffering from severe neurological problems, immunological problems, digestive problems, musculoskeletal problems, fascial problems, neuro-cognitive problems, visual problems, cranial nerve problems, dental problems, fertility issues, chronic pain, and with people who are severely disabled. This version is not the same as the one discovered and studied previously in the twentieth century before it was given the name Lyme disease. Originally it was referred to as a Borrelia infection, causing mild to moderate neurological effects. The two versions are not the same, differing in their gene sequence in some fashion, likely due to purposeful genetic engineering with bad intent.

The Borrelia bacteria has been around for a long time, however, existing perhaps even before the arrival of humanity. The frozen 5300-year-old remains of the Iceman Otzi found in the Austrian Alps in 1991 were tested for the presence of Lyme bacteria. Even though Otzi was an otherwise healthy 40-year-old who met his death at the point of a hostile arrow, he tested positive for Borrelia burgdorferi spirochetes and likely suffered from some of the same symptoms experienced by sufferers today.

Lyme is primarily a neurological disease, even though symptoms vary from brain fog to fatigue to achiness of joints and tissues that are clearly outside the scope of practical neurology. At the core of all manifest symptoms is neuro-inflammation, however, thus the primary use of anti-inflammatory drugs for symptomatic treatment. There are multiple causes for neuro-inflammation, including neurotoxins, head trauma, autoimmunity, aging, vascular, and exposure to microwave and cellular radiation, but the focus of this review will be upon pathogenic influences, namely by Borrelia infection.

(Before continuing, I want to note that in the instance of brain trauma, the body will naturally initiate inflammation in the process of healing the trauma. In head trauma, cells or some component of the brain may be damaged or destroyed, resulting in dead tissue. One theory is that the body allows pathogens into the area to clean up the debris, essentially becoming a feeding ground for microbes. The question remains, however, as to whether the microbes are there to help or there as parasites.)

There are seed-like structures, or cysts or round bodies, found on the outside surface of the spirochetes which contain the complete DNA of the bacteria, like babies that are ready to hatch and proliferate further. When the spirochete is threatened, say by injecting toxic ozone into its environment or by taking an antibiotic, the worm-like structure becomes vulnerable and dies almost immediately. But what doesn't die are these little attached cysts. These round bodies have been isolated and exposed to extreme toxins, and heated with a direct flame, for long periods without impact. They are able to survive anything we can throw at them, remaining intact and ready to hatch into the next generation of damaging spirochetes.

We have no pharmacological treatment that works. With a new infection of Lyme disease, doxycycline is advised for treatment for a period of time to kill the invaders. While immediately effective against the spirochetes, doxycycline does not touch the cysts. Perhaps it may not be possible to completely eradicate Borrelia once infected, but there is one viable strategy in which we can use a bait and switch tactic to minimize their impact.

We can manipulate the micro-environment where the cysts are located and seduce them into hatching out into spirochetes which can be readily killed with antibiotics. The young spirochetes do not have the ability to make these cysts. It takes some time for them to grow these survival capsules, so if you can catch the spirochete soon after it hatches, you can halt the cycle.

The bait that we can use to enhance the micro-environment where they are found is hyaluronic acid. The reason Borrelia is typically found in certain body parts – joints, tendons, tendon sheaths, the fascia, dental ligaments, in the eyes – is because these are areas that are normally high in hyaluronic acid. So, when we supplement with hyaluronic acid, which is taken up by and circulated throughout the body, the cysts detect the increase in a favorable environment and begin to hatch their spirochetes to take advantage of the enhanced body chemistry. If we follow that with an effective microbial agent, then we succeed in killing them and breaking the cycle.

The cysts, for all intents and purposes, are embedded in the body's tissues and lie there in dormancy until the body's chemistry becomes more favorable to hatch their offspring. The cysts do not rob the body of nutrients, so one might imagine they have little impact at all on the body in their latent state. But the body's immune system is not happy about them being there, attacking any tissue where the cysts are embedded. When Lyme disease is treated over long periods with any sort of anti-microbial drug, the bacteria retreat into cystic form, which makes them untouchable. While we cannot kill them in this form, our body's immune system begins an autoimmune assault on our own tissues, resulting in pain and suffering, without any effect against the cause. When such an autoimmune disease is diagnosed, your doctor generally recommends an anti-inflammatory agent to treat the symptoms, again, with no effect on the causative bacteria.

Because antibiotics have been overused over the past century and have been found to be increasingly ineffective, anti-microbials are a more effective measure to destroy this and other bacteria, viruses, and parasites. Use of an anti-microbial to bring about successful closure to the cycle of a Lyme infection is recommended as the final element to the bait and switch tactic mentioned above.

extracted from a YouTube video by Dr. Dietrich Klinghardt on September 2, 2021

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